Whole-body vibration may be as effective as regular exercise

Source: The Endocrine Society

Mouse study is the first to show less strenuous alternative can benefit bone health.

hypervibe 2A less strenuous form of exercise known as whole-body vibration (WBV) can mimic the muscle and bone health benefits of regular exercise in mice, according to a new study published in the Endocrine Society’s journal Endocrinology.

WBV consists of a person sitting, standing or lying on a machine with a vibrating platform. When the machine vibrates, it transmits energy to the body, and muscles contract and relax multiple times during each second.

Many people find it challenging to exercise regularly and that is contributing to the obesity and diabetes epidemics. These disorders can increase the risk of bone fractures. Physical activity can help to decrease this risk and reduce the negative metabolic effects of each condition.

“Our study is the first to show that whole-body vibration may be just as effective as exercise at combating some of the negative consequences of obesity and diabetes,” said the study’s first author, Meghan E. McGee-Lawrence, Ph.D., of Augusta University in Augusta, Ga. “While WBV did not fully address the defects in bone mass of the obese mice in our study, it did increase global bone formation, suggesting longer-term treatments could hold promise for preventing bone loss as well.”

To conduct the study, researchers examined two groups of 5-week-old male mice. One group consisted of normal mice, while the other group was genetically unresponsive to the hormone leptin, which promotes feelings of fullness after eating. Mice from each group were assigned to sedentary, WBV or treadmill exercise conditions.

After a week-long period to grow used to the exercise equipment, the groups of mice began a 12-week exercise program. The mice in the WBV group underwent 20 minutes of WBV at a frequency of 32 Hz with 0.5g acceleration each day. Mice in the treadmill group walked for 45 minutes daily at a slight incline. For comparison, the third group did not exercise. Mice were weighed weekly during the study.

The genetically obese and diabetic mice showed similar metabolic benefits from both WBV and exercising on the treadmill. Obese mice gained less weight after exercise or WBV than obese mice in the sedentary group, although they remained heavier than normal mice. Exercise and WBV also enhanced muscle mass and insulin sensitivity in the genetically obese mice. Although there were no significant effects in the young healthy mice, the low-intensity exercise and WBV protocols were designed for successful completion by obese mice. These findings suggest that WBV may be a useful supplemental therapy to combat metabolic dysfunction in individuals with morbid obesity.

Journal Reference:

  1. Meghan E. McGee-Lawrence Karl H. Wenger Sudipta Misra Catherine L. Davis Norman K. Pollock Mohammed Elsalanty Kehong Ding Carlos M. Isales Mark W. Hamrick Joanna R. Erion Marlena Wosiski-Kuhn Phonepasong Arounleut Mark P. Mattson Roy G. Cutler Jack C. Yu Alexis M. Stranahan. Whole-body Vibration Mimics the Metabolic Effects of Exercise in Male Leptin Receptor Deficient Mice

Genetic Profiling for Personalised Health

fitgenesIt’s true you can’t change your genes but you can affect their expression and influence with the right nutritional, exercise and lifestyle choices.

Fitgenes examines a number of genes in categories and their influence on:

  • Inflammation – our body’s natural defence mechanism. How our body protects itself from DNA damage.
  • Vitamin D Receptors – found in almost all cells, a key influence on bone health, immunity, skin and the nervous system.
  • Methylation and Homocysteine Metabolism – influences the processing of brain chemicals determining emotional wellbeing.
  • Cardiovascular Health – the effectiveness of the system delivering oxygen and nutrients to every cell.
  • Fat and Cholesterol Metabolism – influences healthy weight management, appetite control and satiety.

Genetic Profiling for Personalised Health

Fitgenes is founded on the science of nutrigenomics and an understanding that overall health and wellbeing is determined at a cellular level.

Personalised genetic profiling offers insight in to how our body responds to diet, nutrition, exercise and lifestyle. Knowing how to accurately interpret the results and turn them in to actionable, everyday health and wellbeing is where Fitgenes and our practitioners excel. Fitgenes is founded on the science of nutrigenomics and an understanding that overall health and wellbeing is determined at a cellular level. The DNA contained in our cells send ‘messages’, known as ‘gene expression’, that tells the body how to respond to external influences.

Gene expression

Gene expression is modifiable which means our genes are not our destiny, and DNA damage can be repaired. Through the right profile, interpretation and intervention planning, we can make the right nutritional, exercise and lifestyle choices for long-term health and wellbeing.

A Fitgenes practitioner’s careful interpretation of the genes included in our analysis can provide focused assistance with choosing the right exercise interventions, others on your nutritional interventions and others on your lifestyle choices.

The reports provided are comprehensive and provide a lot of information around the impact of gene expression and guidance around choosing better nutritional choices that match your genes. This is a very targeted and personalised approach to healthcare.

Discover the powerful interaction between genetics, diet and lifestyle to meet your personal health and performance goals.

Fitgenes Health and Wellbeing genetic profile report is an in-depth analysis of the genetic variations that can influence the way the body responds to what we eat, how we exercise and the way we live.

Personalised Genetic Profiling

Personalised genetic profiling offers profound insight into how your body responds to diet, exercise and lifestyle choices. Understanding how to interpret the results of your profile report and convert the information in to programs for actionable, everyday health and wellbeing, is where Fitgenes and our accredited practitioners can offer you the potential to be the best you can be.

Our approach is based on the science of nutrigenomics which looks at the interaction between your genes, nutrition and lifestyle choices, and how these influence the genes’ messages. These messages instruct your body on how it should respond to external influences such as diet and lifestyle choices.

The good news is expression of the genes analysed by Fitgenes is modifiable which means our genes are not our destiny.

Our comprehensive Health and Wellbeing Genetic Profile Report is interpreted by a Fitgenes Accredited Practitioner so they can design a personalised plan to meet individual health goals. Effectively, how your genes are reacting to external factors is analysed and reported on, and a plan to address any negative reactions prepared, based on your unique genetic profile and needs.

Fitgenes profile reports are unique and provide practitioners with a powerful resource to design health plans that are highly personalised. These are not one-size-fits-all plans modified to suit different people.

Carb Choice and Your AMY1 CNV

Fitgenes Australia is a world leader in offering the latest information on the role the AMY 1 gene plays in starch carbohydrate metabolism.  We have developed CarbChoice, a personalised genetic profile report of the AMY1 gene CNV, which determines how effectively you metabolise starch from carbohydrates.

Why should you care about your AMY1 gene?

Scientific studies have shown that variations in the human salivary amylase gene (AMY1) differ based on populations, which have traditionally eaten high starch diets, compared to those who have traditionally eaten low starch diets. Variations within the AMY1 gene influence how well your body can breakdown and process starch, meaning that some people can tolerate these carbohydrates better than others. Starch is the most common carbohydrate included in human diets – however, there is a considerable range of variation between cultures of dietary starch intake. Traditionally, “high starch” populations tended to be agricultural societies and arid region hunter-gathers, while “low starch” populations included rainforest and arctic hunter-gatherers and some pastoralists.

Importance of the AMY1 Gene,  AMY1 is a gene that produces the enzyme ‘amylase’.

What is Amylase?

Amylase is found in our saliva and plays a major role in the digestion of starch, which is a carbohydrate found in grains, legumes, fruit, vegetables, nuts and seeds. Amylase begins the process of carbohydrate metabolism in the mouth.

What will knowing my AMY1 gene copy number variation tell me?

A lower AMY1 copy number indicates:

Test your AMY 1 gene to help you understand:

  • How effectively do you metabolise and tolerate carbohydrates
  • How effectively do you metabolise and tolerate gluten
  • Do carbohydrates put you at an increased risk of being overweight
  • Do carbohydrates put you at an increased risk of having diabetes
  • How many grams of carbohydrates can you eat per day
  • What type of carbohydrates should you avoid
  • How effectively do you use your carbohydrates for energy

Who should test their AMY 1 Gene?

Anyone who wants to understand how their body metabolises and tolerates carbohydrates and make the best dietary choices for themselves.

  • Gluten intolerant or Coeliacs
  • Food sensitivities
  • Struggling to lose weight or maintain weight loss
  • Diabetic or insulin resistant
  • Gut dysbiosis under or over growth
  • Autoimmune issues
  • Persistent infections such as thrush or urinary tract infections
  • Have periodontal (gum) disease

For more information or to book your Fitgenes testing schedule a consultation with Manuela Boyle or Dr Amy Carmichael 07 5522 0505

Vitamin D Deficiency More Widespread

laughing woman in green field

www.enc.org.au

A review into vitamin D supplementation guidelines is being called for after Australia’s largest ever study into vitamin D deficiency found it affects more Australians and lasts longer than previously believed.

The University of Sydney researchers found vitamin D deficiency was not at its worst in winter, as previously thought but in spring.
During a two year period, the prevalence of vitamin D deficiency (>50mM) ranged from 33% in summer to 58% in spring.

The researchers said current testing guidelines did not take seasonal variation into account. “Ideally testing should occur in spring when vitamin D levels reach their lowest concentration” said lead researcher Professor Stephen Boyages, an endocrinologist at Sydney’s Westmead Hospital. “If an individual is found to be deficient, a subsequent test three months afterwards would see if they have been able to replenish their vitamin D.”
“Similarly, use of vitamin D supplements currently fail to address this factor of seasonal variation”, he added.

Being a young female aged 20-39 years of age from a socio-economically disadvantaged background, and being an inpatient, was identified as a new risk factor for vitamin D deficiency.

Urban living was also newly identified s a strong risk factor for vitamin D deficiency, with linear increase in vitamin D levels found with distance from a major city.

The study looked at 24,000 samples from walk-in and inpatients in NSW.

Vitamin D deficiency is implicated in a number of diseases including cancer and diabetes.

Clinical endocrinology 2012; 77:515-523, published in Primary Care Nurse Magazine

 

For those who are struggling to lose weight, an undiagnosed Vitamin D deficiency could be a reason why you are not seeing the results you were hoping for. For more information about the importance of Vitamin D, book an appointment with one of our Doctors. Call us on (07) 5522 0505 to find out more!

What?? Low calorie fake fat makes you….fat?

woman kicking food

By Dr. Karen Coates

Sometimes things just don’t go to plan. A great marketing idea.

A weight loss product, complete with calorie in, calorie out research that, in theory, should make the product perfect for a weight loss diet.

But….then there’s this study published that blows the whole thing out of the water. Feed rats the low fat potato chips and they gain more weight than the rats fed the Real Thing.

Take home message from Dr Karen:

Don’t eat food that has been fiddled with by human hands, especially if it’s been done in a chemical lab rather than a commercial kitchen.

Swithers, S.et al, 2011. Fat Substitutes Promote Weight Gain in Rats Consuming High-Fat Diets. Behavioral Neuroscience, 125 (4).

How to calculate your BMI

DO YOU HAVE A HEALTHY

The formula for calculating BMI uses weight in kilograms and height in metres:

BMI (kg/m2) = Weight (kg) ÷ Height (m) 2.

It can also be calculated using weight in pounds and height in inches:

BMI (lb/in2) = Weight (lb) ÷ Height (in) 2 x 703.

Online calculators that automatically do the computations from an entered height and weight are a convenient method of determining BMI.
Definitions of overweight and obesity

Over 50 healthcare organisations around the world, use the same BMI standards to define adult overweight and obesity.

Definition BMI
Overweight: 25-29.9
Obese: 30-39.9
Morbidly Obese: 40+

One in Four Australians are obese

Obesity-rfrwl

One in four Australians aged 18 years and over were obese in 2007-08, according to figures released today by the Australian Bureau of Statistics.

Since 1995, the rate of obesity has risen from 19% to 24%, with men gaining weight faster than women.

There were just as many people overweight (37%) as there were people of normal weight (37%) in 2007-08, a slight shift from 1995 when there were more people of normal weight (41%) than there were people overweight (38%).

Rates of obesity were related to a number of environmental and socio-economic conditions: A third of Australian adults living in areas of most disadvantage were obese (33%), almost double that of people in areas of least disadvantage (17%). People who had not completed Year 12 were more likely to be obese (31%) than those who had completed this level of education (19%). More adults in outer regional and remote Australia were obese (31%) than those in major cities (23%).

When data on overweight and obesity are combined, the picture of increasing weight gain in Australians becomes more evident. In 2007-08, 61% of adult Australians were overweight or obese. This rate was higher for men (68%) than women (55%), and higher for older people than younger people. Three-quarters of 65-74 year olds were overweight or obese (75%) compared with 37% of 18-24 year olds.

The consequences of this level of overweight and obesity are increased risks of chronic health conditions, increased health service use and increased mortality.

(Source: Australian Bureau of Statistics)